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Acute hyponatremia can cause death if cerebral edema and seizures usually are not treated promptly[1]. Conversely, osmotic demyelination syndrome (ODS) will happen with fast correction of extreme chronic hyponatremia (serum sodium concentration 120 mmol/L or much less) that has been present for much more than 2 or three d, the time necessary for the cerebral adaptation to occur[2]. Excessive correction of chronic hyponatremia triggers a cascade of injury in the brain beginning with breakdown from the blood-brain barrier and culminating within the programmed death of oligodendrocytes, the cells that type myelin sheaths in the central nervous system[3]. Recognized danger aspects for ODS are hyponatremia (each in duration and severity), speedy correction of hyponatremia with additional than 12 mmol/L in less than 24 h, hypokalemia on presentation[4-6], low BUN with malnou.