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A direct link does not seem supported by the current literature.FIGURE 2 Muscle fat determination (by MRI) on 68 participants who consumed either HFCS or sucrose to provide 8, 18, or 30 of power in low-fat milk throughout a 10-wk, free-living trial. Reproduced from (69) with permission. (A) Gluteus maximus. (B) Vastus lateralis. HFCS, higher fructose corn syrup.Does Consumption of Fructose-Containing Sugars Raise Blood PressureSome research have recommended that consumption of added sugars could lead to a rise in blood pressure (83,84). On the other hand, results in human studies are equivocal (85). Some epidemiologic studies have suggested that improved consumption of sugars may possibly increase blood pressure (83, 84,86); nevertheless, other research have not confirmed these findings (85).Johnson et al. (25) have SH5-07 manufacturer proposed a mechanism by way of which increased consumption of sugars could possibly bring about an increase in blood pressure. These investigators propose a model where fructose metabolism inside the liver results in consumption of ATP, which can be eventually degraded to uric acid. Uric acid in turn, as outlined by this model, could result in endothelial dysfunction, top to higher blood pressure. Analysis in our laboratory compared 8, 18, or 30 of calories from HFCS or sucrose for the duration of a 10-wk, free-living period in 352 normotensive men and women and located no enhance in blood pressure (75) (Fig. three). A subsequent study study by our group showed that HFCS consumed at 18 of calories compared with sucrose at 18 of calories, fructose at 9 of calories, or glucose at 9 of calories consumed during a 10-wk period within a free-living cohort of 123 folks did not raise either systolic or diastolic blood stress. Sun et al. (87) looked at nationally representative information comparing fructose consumption with uric acid measurements and did not discover a correlation. These information have been corroborated by analysis in our laboratory showing that at various various dosages of HFCS, sucrose, or fructose, there was no boost in either fasting uric acid or uric AUC (88,89).Endocrine response to sugar-sweetened beveragesFIGURE three Blood pressure response in 352 men and women who consumed either HFCS or sucrose to provide eight, 18, or 30 of calories in low-fat milk during a 10-wk, free-living trial. The white bars indicate baseline measurements plus the black bars indicate measurements obtained following the 10-wk intervention. Adapted with permission from (75). (A) Diastolic blood stress. (B) Systolic blood stress. HFCS, high fructose corn syrup.causes. Several investigators have suggested that enhanced consumption of fructose-containing sugars could result in PubMed ID:http://www.ncbi.nlm.nih.gov/pubmed/20095872 an increase in danger components for MetS. Maersk et al. (26) reported a 6-mo trial of men and women consuming 1 L/d of sucrose-sweetened cola, diet plan cola, milk, or water. They reported that consumption of sucrose-sweetened cola elevated danger components for MetS. Stanhope et al. (16) compared consumption of fructose with glucose at 25 of calories and reported increases in visceral abdominal fat inside the fructose participants. They speculated that this could improve the danger of MetS. Other investigators have challenged these findings. Sun et al. (94) carried out an analysis of nationally representative information and identified no correlation in between fructose-containing sugars and prevalence of MetS. RCTs carried out in our research laboratory compared the effects of 8, 18, or 30 of calories from either HFCS or sucrose on body weight and abdominal fat in a cohort of 116 ind.